Last update: |
||
23-Jan-2017
|
Arch Hellen Med, 34(1), January-February 2017, 127-131 SPECIAL ARTICLE Postprandial dysmetabolism C. Liaskos,1 N. Katsilambros1,2 |
Postprandial dysmetabolism is characterized by an excessive rise in the postprandial serum levels of glucose and triglycerides. It is an independent risk factor for development of cardiovascular disease, even in non-diabetic subjects. Mechanisms which may be involved in the increased incidence of cardiovascular disease are an increase in intracellular oxidative stress, endothelial dysfunction and coagulation disturbances during postprandial hyperglycemia and hypertriglyceridemia. Therapeutic intervention should aim to reduce postprandial glycemia and lipemia, but also fasting levels of glucose and triglycerides. This may be achieved by modification of life-style factors, including changing the type of fatty acids consumed (unsaturated or saturated) and the cooking method, and addition of certain ingredients to the diet. In some cases, appropriate drugs may be needed.
Key words: Hyperglycemia, Hypertriglyceridemia, Postprandial.